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The vasodilatory effects of both flavonoids were inhibited by an Akt inhibitor, highlighting the significance of the Akt pathway

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Instead of differentiating into primitive oocytes as previously hypothesized (given the absence of progeny from transplanted AFSCs), these cells operate by modulating key cellular processes related to follicular atresia, specifically by regulating the expression of genes associated with cell death and survival in GCs, which are crucial for follicular development